Fig. 5From: Propofol postconditioning ameliorates hypoxia/reoxygenation induced H9c2 cell apoptosis and autophagy via upregulating forkhead transcription factors under hyperglycemiaEffects of Silencing of FoxO1 or FoxO3a in the absence or presence of propofol in H9c2 cells subjected to HR (hypoxia/reoxygenation) under hyperglycemia. a Representative image of FoxO1 silence vs normal group. b Representative images of FoxO3a silence vs normal group. c Representative images of FoxO1 silence in HR model. d Representative images of FoxO3a silence in HR model. Data are mean ± SEM from three independent experiments each performed in triplicate. *P < 0.05, **P < 0.01Back to article page